Erythrocytes Induce Vascular Dysfunction in COVID-19 - Mahdi, Ali; Collado, Aida; Tengbom, John; Jiao, Tong; Wodaje, Tigist; Johansson, Niclas; Farnebo, Filip; Färnert, Anna; Yang, Jiangning; Lundberg, Jon O.; Zhou, Zhichao; Pernow, John (JACC. Basic to translational science, 2022)
Keywords: arginase; Cardiovascular; Clinical Research; COVID-19; endothelial dysfunction; nitric oxide; reactive oxygen species; red blood cells; red blood cells; nitric oxide; H-RBC; ACh; C19-RBC; endothelial dysfunction; TNF; arginase; EIR; COVID-19; IFN; EDR; RBC; RHI; SNP; reactive oxygen species; ROS; HNE; red blood cell; sodium nitroprusside; endothelium-dependent relaxation; red blood cell from patients with COVID-19; red blood cell from healthy subjects; hydroxynonenal; interferon; tumor necrosis factor; reactive hyperemia index; acetylcholine; endothelium-independent relaxation; IFN, interferon; SNP, sodium nitroprusside; RBC, red blood cell; H-RBC, red blood cell from healthy subjects; TNF, tumor necrosis factor; EIR, endothelium-independent relaxation; RHI, reactive hyperemia index; ACh, acetylcholine; EDR, endothelium-dependent relaxation; C19-RBC, red blood cell from patients with COVID-19; HNE, hydroxynonenal; ROS, reactive oxygen species