An alternative CTCF isoform antagonizes canonical CTCF occupancy and changes chromatin architecture to promote apoptosis - Li, Jiao; Huang, Kaimeng; Hu, Gongcheng; Babarinde, Isaac A.; Li, Yaoyi; Dong, Xiaotao; Chen, Yu-Sheng; Shang, Liping; Guo, Wenjing; Wang, Junwei; Chen, Zhaoming; Hutchins, Andrew P.; et al. (Nature communications, 2019)
Keywords: 140/58; 38/15; 38/39; 38/89; 38/91; 45/15; 631/208/200; 631/337/100/101; 631/337/1645/1946; 631/337/572/2102; 82/83; Alternative Splicing; Apoptosis; Architecture; Binding; Binding, Competitive; CCCTC-Binding Factor - chemistry; CCCTC-Binding Factor - metabolism; CCCTC-Binding Factor - physiology; Chromatin; Chromatin - chemistry; Chromatin - metabolism; Cohesin; Deactivation; Deoxyribonucleic acid; DNA; Exons; Gene expression; Gene regulation; Genomes; Genomic imprinting; HEK293 Cells; HeLa Cells; Humanities and Social Sciences; Humans; Inactivation; Insulation; Mammals; multidisciplinary; Nucleotide sequence; Occupancy; Protein Isoforms - chemistry; Protein Isoforms - metabolism; Protein Isoforms - physiology; Science; Science (multidisciplinary); X chromosomes; X-chromosome inactivation