STT3-dependent PD-L1 accumulation on cancer stem cells promotes immune evasion - Hsu, Jung-Mao; Xia, Weiya; Hsu, Yi-Hsin; Chan, Li-Chuan; Yu, Wen-Hsuan; Cha, Jong-Ho; Chen, Chun-Te; Liao, Hsin-Wei; Kuo, Chu-Wei; Khoo, Kay-Hooi; Hsu, Jennifer L.; Li, Chia-Wei; et al. (Nature communications, 2018)
Keywords: 13/1; 14/19; 631/67; 631/80/458/1524; 82/29; 96/106; 96/35; Animals; B7-H1 Antigen - genetics; B7-H1 Antigen - immunology; beta Catenin - genetics; beta Catenin - immunology; Cancer; Cancer immunotherapy; Cell Line, Tumor; DNA Topoisomerases, Type II - genetics; DNA Topoisomerases, Type II - immunology; Enrichment; Epithelial-Mesenchymal Transition; Etoposide; Female; Gene Expression Regulation, Neoplastic; Glycosylation; Glycosyltransferase; Hexosyltransferases - genetics; Hexosyltransferases - immunology; Humanities and Social Sciences; Humans; Immune Evasion; Immunotherapy; Membrane Proteins - genetics; Membrane Proteins - immunology; Mesenchyme; Mice, Inbred BALB C; Mice, Knockout; multidisciplinary; Neoplasms - genetics; Neoplasms - immunology; Neoplasms - physiopathology; Neoplastic Stem Cells - cytology; Neoplastic Stem Cells - immunology; PD-L1 protein; Poly-ADP-Ribose Binding Proteins - genetics; Poly-ADP-Ribose Binding Proteins - immunology; Science; Science (multidisciplinary); Stem cells; Transcription; β-Catenin