Parkin regulation of CHOP modulates susceptibility to cardiac endoplasmic reticulum stress - Han, Kim; Hassanzadeh, Shahin; Singh, Komudi; Menazza, Sara; Nguyen, Tiffany T.; Stevens, Mark V.; Nguyen, An; San, Hong; Anderson, Stasia A.; Lin, Yongshun; Zou, Jizhong; Murphy, Elizabeth; et al. (Scientific reports, 2017)
Keywords: 13/106; 13/89; 59/57; 631/337; 64/60; 692/4019/592/1540; 82/29; 82/80; 96/31; Animals; Aorta; Apoptosis; Cardiomyocytes; Cardiomyopathy; Cardiomyopathy, Dilated - metabolism; Cardiomyopathy, Dilated - pathology; CCAAT/enhancer-binding protein; Cell death; Cell Line; Cell Line, Tumor; Dilated cardiomyopathy; Endoplasmic reticulum; Endoplasmic Reticulum Stress; Heart; Heart diseases; HEK293 Cells; Humanities and Social Sciences; Humans; Induced Pluripotent Stem Cells - cytology; Induced Pluripotent Stem Cells - metabolism; Male; Mice; Mice, Inbred C57BL; Mortality; multidisciplinary; Myocardium; Myocytes, Cardiac - cytology; Myocytes, Cardiac - metabolism; Parkin protein; Post-translation; Proteasomes; Rodents; Science; Science (multidisciplinary); Transcription Factor CHOP - metabolism; Tunicamycin; Ubiquitin; Ubiquitin-protein ligase; Ubiquitin-Protein Ligases - genetics; Ubiquitin-Protein Ligases - metabolism; Ventricle; Ventricular Remodeling