Galectin-9 restricts hepatitis B virus replication via p62/SQSTM1-mediated selective autophagy of viral core proteins - Miyakawa, Kei; Nishi, Mayuko; Ogawa, Michinaga; Matsunaga, Satoko; Sugiyama, Masaya; Nishitsuji, Hironori; Kimura, Hirokazu; Ohnishi, Makoto; Watashi, Koichi; Shimotohno, Kunitada; Wakita, Takaji; Ryo, Akihide (Nature communications, 2022)
Keywords: 14/19; 14/28; 631/326/596; 631/326/88; 631/80/39; 82/47; Antiviral Agents - pharmacology; Antiviral drugs; Autophagosomes - metabolism; Autophagy; Autophagy - drug effects; Core protein; Crosstalk; Degradation; Ectopic expression; Galectin-9; Galectins - genetics; Galectins - metabolism; Galectins - pharmacology; Gene Expression; HEK293 Cells; Hep G2 Cells; Hepatitis; Hepatitis B; Hepatitis B virus - drug effects; Hepatitis B virus - metabolism; Hepatocytes; Humanities and Social Sciences; Humans; Immune response; Immune system; Innate immunity; Interferon; Macroautophagy - drug effects; multidisciplinary; Pathogens; Phagosomes; Proteins; Proteolysis; Replication; Science; Science (multidisciplinary); Sequestosome-1 Protein - genetics; Sequestosome-1 Protein - metabolism; Ubiquitination; Viral Core Proteins - metabolism; Viral infections; Virus Replication - drug effects