TDAG8, TRPV1, and ASIC3 involved in establishing hyperalgesic priming in experimental rheumatoid arthritis - Hsieh, Wei-Shan; Kung, Chia-Chi; Huang, Shir-Ly; Lin, Shih-Chang; Sun, Wei-Hsin (Scientific reports, 2017)
Keywords: 13/51; 42/89; 631/378/1959/2605; 64/60; 692/699/1670/498; Acid Sensing Ion Channels - genetics; Acid Sensing Ion Channels - metabolism; Acidosis; Animal models; Animals; Antigens, CD - metabolism; Antigens, Differentiation, Myelomonocytic - metabolism; Arthralgia - etiology; Arthralgia - physiopathology; Arthritis, Experimental; Arthritis, Rheumatoid - complications; Arthritis, Rheumatoid - genetics; Arthritis, Rheumatoid - pathology; Autoimmune diseases; Biomarkers; Capsaicin receptors; Cell death; Chronic pain; Cytokines - blood; Cytokines - metabolism; Disease Models, Animal; Female; Gene Expression; Humanities and Social Sciences; Hydrogen ion concentration; Hydrogen ions; Hyperalgesia - etiology; Hyperalgesia - physiopathology; Immunohistochemistry; Joint diseases; Lymphocytes T; Macrophages - immunology; Macrophages - metabolism; Macrophages - pathology; Male; Mice; Mice, Knockout; multidisciplinary; Pain; Receptors, G-Protein-Coupled - genetics; Receptors, G-Protein-Coupled - metabolism; Rheumatoid arthritis; Science; Science (multidisciplinary); Synovial fluid; Transient receptor potential proteins; TRPV Cation Channels - genetics; TRPV Cation Channels - metabolism