Increased plasmablasts enhance T cell-mediated beta cell destruction and promote the development of type 1 diabetes - Ling, Qing; Shen, Lei; Zhang, Wei; Qu, DuoDuo; Wang, Hongdong; Wang, Bin; Liu, Yong; Lu, Jing; Zhu, Dalong; Bi, Yan (Molecular medicine (Cambridge, Mass.), 2022)
Keywords: Animals; Antibodies; Antigens; Autoimmunity; B cell subset; Biomarkers; Case-Control Studies; Cells; Diabetes; Diabetes Mellitus, Type 1 - diagnosis; Diabetes Mellitus, Type 1 - etiology; Diabetes Mellitus, Type 1 - metabolism; Disease; Disease Models, Animal; Disease Susceptibility - immunology; Female; Flow cytometry; Humans; Immune response; Immunohistochemistry; Immunophenotyping; Immunotherapy; Insulin-Secreting Cells - immunology; Insulin-Secreting Cells - metabolism; Insulin-Secreting Cells - pathology; Ketosis; Lymphatic system; Lymphocyte Activation; Lymphocytes; Male; Mice; Mice, SCID; Pathogenesis; Peptides; Plasmablast; T cell; T-Lymphocytes - immunology; T-Lymphocytes - metabolism; Type 1 diabetes; United States--US; Autoimmunity; B cell subset; Plasmablast; Type 1 diabetes; T cell