Treatment with the anti-IL-6 receptor antibody attenuates muscular dystrophy via promoting skeletal muscle regeneration in dystrophin-/utrophin-deficient mice - Wada, Eiji; Tanihata, Jun; Iwamura, Akira; Takeda, Shin’ichi; Hayashi, Yukiko K.; Matsuda, Ryoichi (Skeletal muscle, 2017)
Keywords: Animal models; Arthritis; Cardiac muscle; Care and treatment; Creatine; Creatine kinase; Cytokines; Degeneration; Diaphragm; Disease; Drug dosages; Duchenne muscular dystrophy; Duchenne's muscular dystrophy; Dystrophin; Embryos; Fibrosis; Health aspects; Immunoglobulin G; Inflammation; Inflammatory diseases; Interleukin 6; Interleukin 6 receptors; Kyphosis; Metabolism; Muscle regeneration; Muscular dystrophy; Musculoskeletal system; Myosin; Pathology; Physiological aspects; Quadriceps muscle; Rats; Rodents; Skeletal muscle; STAT3; Stem cells; Tissue engineering; Transcription; Transgenic animals; Utrophin; Vertebrae; Duchenne muscular dystrophy; Muscle regeneration; Fibrosis; STAT3; Interleukin-6