BabaoDan attenuates high-fat diet-induced non-alcoholic fatty liver disease via activation of AMPK signaling - Sheng, Dandan; Zhao, Shanmin; Gao, Lu; Zheng, Huifei; Liu, Wenting; Hou, Jing; Jin, Yuxiang; Ye, Fei; Zhao, Qiudong; Li, Rong; Zhao, Naping; Zhang, Li; et al. (Cell & bioscience, 2019)
Keywords: Adipose tissue; Alanine; Alanine transaminase; AMPK pathway; Antigens; Asian medicine; Aspartate aminotransferase; Babaodan; Biological products; Carbohydrates; CD36 antigen; Cholesterol; Cholesterol tests; Complications and side effects; Diet; Diet therapy; Experiments; Fatty liver; Genes; Glucose; High density lipoprotein; High fat diet; Interleukin 6; Kidneys; Laboratory animals; Leptin; Lipid metabolism; Liver cancer; Liver diseases; Low density lipoprotein; Low density lipoproteins; Medical research; Non-alcoholic fatty liver disease; Obesity; Oxidation; Oxidation-reduction reactions; Polymerase chain reaction; Proteins; Risk factors; Serum levels; Traditional Chinese medicine; Triglycerides; Tumor necrosis factor-TNF; Tumor necrosis factor-α; China; United Kingdom--UK; United States--US; Japan; Lipid metabolism; AMPK pathway; Non-alcoholic fatty liver disease; Babaodan