TLR4 activation by lipopolysaccharide confers survival advantage to growth factor deprived prostate cancer cells - Jain, Sumeet; Suklabaidya, Sujit; Das, Biswajit; Raghav, Sunil K.; Batra, Surinder K.; Senapati, Shantibhusan (The Prostate, 2015)
Keywords: Animals; Apoptosis; Apoptosis - drug effects; Autophagy; Autophagy - drug effects; CCL2; Cell death; Cell Line, Tumor; Cell Survival - drug effects; Cell Survival - physiology; Cellular Senescence - drug effects; Chemokine CCL2 - analysis; Culture Media, Serum-Free; Escherichia coli; Humans; infection; Intercellular Signaling Peptides and Proteins - administration & dosage; lipopolysaccharide; Lipopolysaccharides; Lipopolysaccharides - pharmacology; LPS; Macrophages; Male; metabolic stress; Molecular modelling; Monocyte chemoattractant protein 1; Neoplasm Transplantation; Neoplasms, Experimental; Phagocytosis; Prostate cancer; Prostatic Neoplasms - chemistry; Prostatic Neoplasms - pathology; Rats; RNA, Messenger - analysis; RNA, Small Interfering - pharmacology; Senescence; Starvation; Survival factor; TLR4; TLR4 protein; Toll-Like Receptor 4 - antagonists & inhibitors; Toll-Like Receptor 4 - genetics; Toll-Like Receptor 4 - physiology; Toll-like receptors; Tumors; infection; prostate cancer; autophagy; CCL2; metabolic stress; lipopolysaccharide; TLR4; LPS