miR-589 promotes gastric cancer aggressiveness by a LIFR-PI3K/AKT-c-Jun regulatory feedback loop - Zhang, Feifei; Li, Kaitao; Pan, Mingxin; Li, Weidong; Wu, Juan; Li, Mingyi; Zhao, Liang; Wang, Hui (Journal of experimental & clinical cancer research, 2018)
Keywords: Analysis; Animals; Breast cancer; Cell growth; Cell Line, Tumor; Cytokines; Development and progression; DNA methylation; Experiments; Feedback; Feedback loop; Female; Gastric cancer; Gene expression; Genetic aspects; Health aspects; Humans; Kinases; Laboratories; Leukemia; LIFR; Liver cancer; Male; Medical prognosis; Metastasis; Mice; Mice, Nude; MicroRNA; MicroRNAs; MicroRNAs - metabolism; miR-589; Neoplasm Metastasis; Phosphatidylinositol 3-Kinases - metabolism; Physiological aspects; Proteins; Proto-Oncogene Proteins c-akt - metabolism; Stomach cancer; Stomach Neoplasms - genetics; Stomach Neoplasms - pathology; Tumorigenesis; China; United States--US; Feedback loop; LIFR; miR-589; Metastasis; Gastric cancer