Lack of clinical therapeutic benefit of antidepressants is associated overall activation of the inflammatory system - Carvalho, L.A.; Torre, J.P.; Papadopoulos, A.S.; Poon, L.; Juruena, M.F.; Markopoulou, K.; Cleare, A.J.; Pariante, C.M. (Journal of affective disorders, 2013)
Keywords: Admissions; Adult; Adult and adolescent clinical studies; Affective disorders; Antidepressant agents; Antidepressant drugs; Antidepressive Agents - adverse effects; Biological and medical sciences; Biological markers; Case-Control Studies; Cortisol; Cytokines; Cytokines - blood; Depression; Depression - blood; Depression - drug therapy; Endophenotype; Female; Hospitalization; Humans; Hydrocortisone - blood; Inflammation - chemically induced; Interleukin-10 - blood; Interleukin-4 - blood; Interleukin-6 - blood; Male; Medical sciences; Medical treatment; Middle Aged; Mood disorders; Neuroinflammation; Neuropharmacology; Pharmacology. Drug treatments; Psychiatry; Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer; Psychoanaleptics: cns stimulant, antidepressant agent, nootropic agent, mood stabilizer..., (alzheimer disease); Psychology. Psychoanalysis. Psychiatry; Psychoneuroimmunology; Psychopathology. Psychiatry; Psychopharmacology; Treatment Outcome; Vascular Endothelial Growth Factor A - blood; Neuroinflammation; Psychoneuroimmunology; Cytokines; Antidepressant agents; Biological markers; Endophenotype; Psychotropic; Cytokine; Biological marker; Pharmacotherapy; Activation; Inflammation; Treatment; Antidepressant agent