A switch from canonical to noncanonical autophagy shapes B cell responses - Martinez-Martin, Nuria; Maldonado, Paula; Gasparrini, Francesca; Frederico, Bruno; Aggarwal, Shweta; Gaya, Mauro; Tsui, Carlson; Burbage, Marianne; Keppler, Selina Jessica; Montaner, Beatriz; Jefferies, Harold B. J.; Nair, Usha; et al. (Science (American Association for the Advancement of Science), 2017)
Keywords: Ablation; Activation; Animals; Autophagy; Autophagy - immunology; B-Lymphocytes - immunology; B-Lymphocytes - virology; Cell activation; Cell culture; Cell differentiation; Computer memory; Differentiation; Differentiation (biology); Down-Regulation; Genetics; Germinal Center - immunology; Germinal Center - virology; Germinal centers; Homeostasis; Imaging; Immune system; Immunological memory; Individualized Instruction; Infections; Lymphocyte Activation; Lymphocyte receptors; Lymphocytes B; Maintenance; Mechanistic Target of Rapamycin Complex 1; Medical innovations; Memory cells; Mice; Mice, Inbred C57BL; Mice, Knockout; Microtubule-Associated Proteins - genetics; Microtubule-Associated Proteins - metabolism; Mitochondria - metabolism; Multiprotein Complexes - metabolism; Pathways; Pharmacology; Rapamycin; Stimulation; Switches; T cell receptors; TOR protein; TOR Serine-Threonine Kinases - metabolism; Viral infections; Virus Diseases - immunology; WD40 Repeats - genetics