Mitochondrial MsrB2 serves as a switch and transducer for mitophagy - Lee, Seung Hee; Lee, Suho; Du, Jing; Jain, Kanika; Ding, Min; Kadado, Anis J; Atteya, Gourg; Jaji, Zainab; Tyagi, Tarun; Kim, Won‐ho; Herzog, Raimund I; Patel, Amar; et al. (EMBO molecular medicine, 2019)
Keywords: Animals; Apoptosis; Autophagy; Blood Platelets - enzymology; Blood Platelets - pathology; Cell Line; Data analysis; Diabetes; Diabetes mellitus; Diabetes Mellitus - blood; Diabetes Mellitus - genetics; Diabetes Mellitus - pathology; EMBO21; EMBO27; Experiments; Female; Gene regulation; Heart; Humans; Insulin; Matrix protein; Methionine; methionine sulfoxide reductase; Methionine Sulfoxide Reductases - blood; Methionine Sulfoxide Reductases - deficiency; Methionine Sulfoxide Reductases - genetics; Mice, Inbred C57BL; Mice, Knockout; Microfilament Proteins - blood; Microfilament Proteins - deficiency; Microfilament Proteins - genetics; Microscopy; Microtubule-Associated Proteins - blood; Mitochondria; Mitochondria - enzymology; Mitochondria - pathology; Mitochondrial Membrane Transport Proteins - metabolism; Mitochondrial Permeability Transition Pore; Mitophagy; Movement disorders; Mutation; Neurodegenerative diseases; Oxidation-Reduction; Oxidative Stress; Parkin protein; Parkinson Disease - blood; Parkinson Disease - genetics; Parkinson Disease - pathology; Parkinson's disease; Platelets; Proteins; Reactive oxygen species; Signal Transduction; Software; Transcription; Ubiquitin-Protein Ligases - blood; Ubiquitin-Protein Ligases - genetics; Ubiquitination; La Jolla California; United States--US; apoptosis; mitophagy; diabetes mellitus; methionine sulfoxide reductase; platelets