miR‐207 Suppresses the Progression of SiO 2 ‐Induced Pulmonary Fibrosis by Targeting Smad3 to Regulate the TGF‐β1/Smad3 Signaling Pathway in C57BL/6 Mice - Zhao, Jia‐hui; Li, Shuang; Du, Shu‐ling; Han, Gui‐zhi; Li, Huan; Shao, Bo; Liu, Xia; Zhou, Yuting; Zhang, Zhao‐qiang (Journal of biochemical and molecular toxicology, 2025)
Keywords: Animals; Disease Progression; Male; Mice; Mice, Inbred C57BL; MicroRNAs - genetics; MicroRNAs - metabolism; Pulmonary Fibrosis - chemically induced; Pulmonary Fibrosis - genetics; Pulmonary Fibrosis - metabolism; Pulmonary Fibrosis - pathology; Pulmonary Fibrosis - prevention & control; Signal Transduction - drug effects; Silicon Dioxide - toxicity; Silicosis - metabolism; Silicosis - pathology; Smad3 Protein - genetics; Smad3 Protein - metabolism; Transforming Growth Factor beta1 - genetics; Transforming Growth Factor beta1 - metabolism; Fn; SiO2‐induced pulmonary fibrosis; α‐SMA; miR‐207; TGF‐β1/Smad3 signaling pathway