CD5L/AIM Regulates Lipid Biosynthesis and Restrains Th17 Cell Pathogenicity - Wang, Chao; Yosef, Nir; Gaublomme, Jellert; Wu, Chuan; Lee, Youjin; Clish, Clary B.; Kaminski, Jim; Xiao, Sheng; Zu Horste, Gerd Meyer; Pawlak, Mathias; Kishi, Yasuhiro; Joller, Nicole; et al. (Cell, 2015)
Keywords: Animals; Apoptosis Regulatory Proteins - metabolism; autoimmunity; biosynthesis; Cell Differentiation; Central Nervous System - pathology; cholesterol; Cholesterol - biosynthesis; Encephalomyelitis, Autoimmune, Experimental - immunology; Encephalomyelitis, Autoimmune, Experimental - pathology; fatty acid composition; Fatty Acids, Unsaturated - metabolism; homeostasis; Humans; ligands; Lipid Metabolism; Lymph Nodes - pathology; Mice; Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism; pathogenicity; Receptor Tyrosine Kinase-like Orphan Receptors - metabolism; Receptors, Immunologic - metabolism; Receptors, Scavenger; sequence analysis; Single-Cell Analysis; Th17 Cells - immunology; Th17 Cells - pathology; transcription factors