The role of S100B/RAGE-enhanced ADAM17 activation in endothelial glycocalyx shedding after traumatic brain injury - Zou, Zhimin; Li, Li; Li, Qin; Zhao, Peng; Zhang, Kun; Liu, Chengyong; Cai, Daozhang; Maegele, Marc; Gu, Zhengtao; Huang, Qiaobing (Journal of neuroinflammation, 2022)
Keywords: ADAM17; ADAM17 Protein - metabolism; Animal models; Animals; Astrocytes; Blood-brain barrier; Blood-Brain Barrier - metabolism; Brain; Brain Injuries, Traumatic - metabolism; Capillary Permeability; Endothelial cells; Endothelial Cells - metabolism; Endothelial glycocalyx; Glycocalyx; Glycocalyx - metabolism; Health aspects; Immunofluorescence; Injuries; Laboratory animals; Lungs; Pathogens; Permeability; Rats; Rodents; S100 Calcium Binding Protein beta Subunit - metabolism; S100b protein; S100B/RAGE; Secondary injury; Signal transduction; Syndecan; Trauma; Traumatic brain injury; Tumor necrosis factor; Tumor necrosis factor-TNF; China; United States--US; S100B/RAGE; Secondary injury; Endothelial glycocalyx; ADAM17; Traumatic brain injury; Blood–brain barrier