Aortic valve replacement in a 41-year-old woman with uncorrected tetralogy of Fallot, pulmonary atresia, and major aortopulmonary collateral arteries: a case report

Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. Case presenta...

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Published inJA clinical reports Vol. 9; no. 1; p. 86
Main Authors Saito, Kazutomo, Iwasaki, Yudai, Tasaki, Takahiro, Saito, Hidehisa, Toyama, Hiroaki, Ejima, Yutaka, Yamauchi, Masanori
Format Journal Article
LanguageEnglish
Published Berlin/Heidelberg Springer Berlin Heidelberg 06.12.2023
Springer Nature B.V
SpringerOpen
Subjects
Anesthesiology
Antibiotics
Aortic valve replacement
Blood
Cardiovascular disease
Case Report
Case reports
Catheters
Congenital diseases
Coronary vessels
Critical Care Medicine
Emergency Medicine
Endocarditis
General anesthesia
Heart rate
Heart surgery
Hemoglobin
Hospitals
Infectious endocarditis
Intensive
Major aortopulmonary collateral arteries
Medicine
Medicine & Public Health
Oxygen saturation
Pain Medicine
Patients
Pulmonary arteries
Tomography
Uncorrected tetralogy of Fallot
Vegetation
Veins & arteries
Weaning
Uncorrected tetralogy of Fallot
Aortic valve replacement
Infectious endocarditis
Major aortopulmonary collateral arteries
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Abstract Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. Case presentation A 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful. Conclusion An adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
AbstractList Abstract Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. Case presentation A 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful. Conclusion An adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
BackgroundTetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare.Case presentationA 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful.ConclusionAn adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. A 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful. An adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
Abstract Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. Case presentation A 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful. Conclusion An adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during childhood, cardiac surgery under cardiopulmonary bypass (CPB) for adult survivors with unrepaired TOF is exceedingly rare. Case presentation A 41-year-old woman with unrepaired TOF, pulmonary atresia (PA), and major aortopulmonary collateral arteries (MAPCAs) developed acute infectious endocarditis (IE). As vegetation gradually increased despite intravenous antibiotic administration, she was scheduled for urgent aortic valve replacement under CPB. Pulmonary blood flow was primarily provided by the MAPCAs originating from the descending aorta. Intra-aortic balloon occlusion for MAPCAs was performed to ensure a bloodless surgical field. Aortic valve replacement was successful. Conclusion An adult with uncorrected TOF developed acute IE and subsequently had successful cardiac surgery under CPB. Understanding TOF physiology with PA and MAPCAs, particularly pulmonary blood flow through MAPCAs, is crucial.
ArticleNumber 86
Author Tasaki, Takahiro
Saito, Kazutomo
Yamauchi, Masanori
Ejima, Yutaka
Iwasaki, Yudai
Saito, Hidehisa
Toyama, Hiroaki
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BackLink https://www.ncbi.nlm.nih.gov/pubmed/38055085$$D View this record in MEDLINE/PubMed
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10.1016/S0735-1097(85)80223-0
10.1056/NEJMicm1912128
10.1016/j.athoracsur.2013.03.070
10.4314/mmj.v34i3.9
10.1016/0735-1097(94)00364-V
10.1016/j.hlc.2008.10.017
10.1053/j.jvca.2018.03.035
10.1016/j.athoracsur.2018.11.016
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Keywords Uncorrected tetralogy of Fallot
Aortic valve replacement
Infectious endocarditis
Major aortopulmonary collateral arteries
Language English
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Snippet Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair...
Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair during...
Abstract Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical...
BackgroundTetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair...
BACKGROUNDTetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical repair...
Abstract Background Tetralogy of Fallot (TOF) is a complex cyanotic congenital heart disease. As most patients with TOF undergo palliative or radical surgical...
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StartPage 86
SubjectTerms Anesthesiology
Antibiotics
Aortic valve replacement
Blood
Cardiovascular disease
Case Report
Case reports
Catheters
Congenital diseases
Coronary vessels
Critical Care Medicine
Emergency Medicine
Endocarditis
General anesthesia
Heart rate
Heart surgery
Hemoglobin
Hospitals
Infectious endocarditis
Intensive
Major aortopulmonary collateral arteries
Medicine
Medicine & Public Health
Oxygen saturation
Pain Medicine
Patients
Pulmonary arteries
Tomography
Uncorrected tetralogy of Fallot
Vegetation
Veins & arteries
Weaning
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Title Aortic valve replacement in a 41-year-old woman with uncorrected tetralogy of Fallot, pulmonary atresia, and major aortopulmonary collateral arteries: a case report
URI https://link.springer.com/article/10.1186/s40981-023-00674-0
https://www.ncbi.nlm.nih.gov/pubmed/38055085
https://www.proquest.com/docview/2898759906/abstract/
https://search.proquest.com/docview/2898954839
https://doaj.org/article/6c9d6aa2fe7c47ff83317a16dd27552e
Volume 9
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