Phosphorylation of FOXP3 controls regulatory T cell function and is inhibited by TNF-α in rheumatoid arthritis - Nie, Hong; Zheng, Yingxia; Li, Runsheng; Guo, Taylor B; He, Dongyi; Fang, Lei; Liu, Xuebin; Xiao, Lianbo; Chen, Xi; Wan, Bing; Chin, Y Eugene; Zhang, Jingwu Z (Nature medicine, 2013)
Keywords: 631/250/127/1220; 631/250/1619/554/1898/1271; 631/250/249/1313/498; 631/250/516; Arthritis, Rheumatoid - immunology; Arthritis, Rheumatoid - metabolism; Biomedicine; Cancer Research; Cells, Cultured; DNA-Binding Proteins - metabolism; Forkhead Transcription Factors - chemistry; Forkhead Transcription Factors - metabolism; Genetic aspects; Health aspects; Humans; Infectious Diseases; Interferon-gamma - biosynthesis; Interleukin-17 - biosynthesis; Interleukin-2 Receptor alpha Subunit - biosynthesis; Interleukin-7 Receptor alpha Subunit - biosynthesis; Metabolic Diseases; Molecular Medicine; Neurosciences; Phosphorylation; Physiological aspects; Protein Phosphatase 1 - metabolism; Rheumatoid arthritis; Risk factors; RNA Interference; RNA, Small Interfering; Synovial Membrane - immunology; Synovial Membrane - metabolism; T cells; T-Lymphocytes, Regulatory - immunology; T-Lymphocytes, Regulatory - metabolism; Th1 Cells - immunology; Th1 Cells - metabolism; Th17 Cells - immunology; Th17 Cells - metabolism; Tumor necrosis factor; Tumor Necrosis Factor-alpha - immunology; Tumor Necrosis Factor-alpha - metabolism; China