APA (7th ed.) Citation

Lakshmanan, A. P., Harima, M., Suzuki, K., Soetikno, V., Nagata, M., Nakamura, T., . . . Watanabe, K. (2013). The hyperglycemia stimulated myocardial endoplasmic reticulum (ER) stress contributes to diabetic cardiomyopathy in the transgenic non-obese type 2 diabetic rats: A differential role of unfolded protein response (UPR) signaling proteins. The international journal of biochemistry & cell biology, 45(2), 438-447. https://doi.org/10.1016/j.biocel.2012.09.017

Chicago Style (17th ed.) Citation

Lakshmanan, Arun Prasath, et al. "The Hyperglycemia Stimulated Myocardial Endoplasmic Reticulum (ER) Stress Contributes to Diabetic Cardiomyopathy in the Transgenic Non-obese Type 2 Diabetic Rats: A Differential Role of Unfolded Protein Response (UPR) Signaling Proteins." The International Journal of Biochemistry & Cell Biology 45, no. 2 (2013): 438-447. https://doi.org/10.1016/j.biocel.2012.09.017.

MLA (9th ed.) Citation

Lakshmanan, Arun Prasath, et al. "The Hyperglycemia Stimulated Myocardial Endoplasmic Reticulum (ER) Stress Contributes to Diabetic Cardiomyopathy in the Transgenic Non-obese Type 2 Diabetic Rats: A Differential Role of Unfolded Protein Response (UPR) Signaling Proteins." The International Journal of Biochemistry & Cell Biology, vol. 45, no. 2, 2013, pp. 438-447, https://doi.org/10.1016/j.biocel.2012.09.017.