Therapeutic role of miR-26a on cardiorenal injury in a mice model of angiotensin-II induced chronic kidney disease through inhibition of LIMS1/ILK pathway - Ni, Weijie; Zhao, Yajie; Shen, Jinxin; Yin, Qing; Wang, Yao; Li, Zuolin; Tang, Taotao; Wen, Yi; Zhang, Yilin; Jiang, Wei; Jiang, Liangyunzi; Wei, Jinxuan; et al. (Chinese medical journal, 2025)
Keywords: ACE inhibitors; Adenoviruses; Angiotensin II - toxicity; Animals; Antibodies; Blood pressure; Cell Line; Creatinine; Cytokines; Disease Models, Animal; Heart; Histology; Humans; Hybridization; Immunohistochemistry; Inflammation; Kidney diseases; Kinases; LIM Domain Proteins - genetics; LIM Domain Proteins - metabolism; Male; Mice; Mice, Inbred C57BL; Mice, Knockout; MicroRNAs; MicroRNAs - genetics; MicroRNAs - metabolism; Original; Peptides; Proteins; Renal Insufficiency, Chronic - chemically induced; Renal Insufficiency, Chronic - genetics; Renal Insufficiency, Chronic - metabolism; Signal Transduction - genetics; Statistical analysis; Variance analysis; United States--US; China; California; Chronic kidney disease; LIMS1 protein; Inflammation; microRNA-26a; Cardiorenal injury; Fibrosis