LncRNA UCA1 promotes keratinocyte-driven inflammation via suppressing METTL14 and activating the HIF-1α/NF-κB axis in psoriasis - Hu, Yibo; Lei, Li; Jiang, Ling; Zeng, Hongliang; Zhang, Yushan; Fu, Chuhan; Guo, Haoran; Dong, Yumeng; Ouyang, Yujie; Zhang, Xiaolin; Huang, Jinhua; Zeng, Qinghai; et al. (Cell death & disease, 2023)
Keywords: 13/1; 13/109; 13/21; 13/51; 13/89; 14/32; 14/34; 45/90; 45/91; 631/250/262/2106/2518; 631/337/176/2016; 692/699/249/1313/1758; 82/58; Antibodies; Biochemistry; Biomedical and Life Sciences; Cell Biology; Cell Culture; Cell Proliferation; Cytokines; Cytokines - metabolism; Endothelial cells; Endothelial Cells - metabolism; Gene expression; Humans; Hypoxia-inducible factor 1a; Immunology; Inflammation; Inflammation - genetics; Inflammation - metabolism; Innate immunity; Keratinocytes; Keratinocytes - metabolism; Life Sciences; Methyltransferase; Methyltransferases - metabolism; Molecular modelling; N6-methyladenosine; NF-kappa B - genetics; NF-kappa B - metabolism; NF-κB protein; Non-coding RNA; Proteomes; Psoriasis; Psoriasis - pathology; RNA, Long Noncoding - genetics; RNA, Long Noncoding - metabolism; Signal transduction; Stat3 protein; Transcriptomes