Interplay of FXN expression and lipolysis in white adipocytes plays a critical role in insulin sensitivity in Friedreich’s ataxia mouse model - Wu, Lin; Huang, Fei; Yang, Lu; Yang, Liu; Sun, Zichen; Zhang, Jinghua; Xia, Siyu; Zhao, Hongting; Ding, Yibing; Bian, Dezhi; Li, Kuanyu (Scientific reports, 2024)
Keywords: 631/443/319; 631/80/304; Adipocytes; Adipocytes, White - metabolism; Adipocytes, White - pathology; Adipose tissue; Age; Animals; Ataxia; Diabetes mellitus; Disease Models, Animal; Forskolin; Frataxin; Friedreich Ataxia - genetics; Friedreich Ataxia - metabolism; Friedreich Ataxia - pathology; Friedreich's ataxia; Homeostasis; Humanities and Social Sciences; Humans; Hyperglycemia; Hypertrophy; Insulin Resistance; Insulin sensitivity; Iron-Binding Proteins - genetics; Iron-Binding Proteins - metabolism; Lipase; Lipase - genetics; Lipase - metabolism; Lipogenesis; Lipolysis; Male; Metabolism; Mice; multidisciplinary; Neurodegenerative diseases; Pioglitazone; PPARγ; Science; Science (multidisciplinary); Sulfur; Susceptibility; Adipose tissue; Frataxin; Insulin sensitivity; PPARγ; Lipolysis