Procyanidin A1 Alleviates Inflammatory Response induced by LPS through NF-κB, MAPK, and Nrf2/HO-1 Pathways in RAW264.7 cells - Han, Shan; Gao, Hongwei; Chen, Shaoru; Wang, Qinqin; Li, Xinxing; Du, Li-Jun; Li, Jun; Luo, Ying-Ying; Li, Jun-Xiu; Zhao, Li-Chun; Feng, Jianfang; Yang, Shilin (Scientific reports, 2019)
Keywords: 13/1; 13/21; 13/95; 692/308/153; 692/4017; Animals; Anti-inflammatory agents; Catechin - chemistry; Catechin - pharmacology; Cell Nucleus - drug effects; Cell Nucleus - metabolism; Cytokines - metabolism; Heme Oxygenase-1 - metabolism; Humanities and Social Sciences; Inflammation - pathology; Inflammation Mediators - metabolism; Inflammatory diseases; Interleukin 6; Lipopolysaccharides; MAP kinase; Membrane potential; Membrane Potential, Mitochondrial - drug effects; Mice; Mitochondria; Mitogen-Activated Protein Kinases - metabolism; Models, Biological; Molecular modelling; multidisciplinary; NF-E2-Related Factor 2 - metabolism; NF-kappa B - metabolism; NF-κB protein; Nitric-oxide synthase; Nuclear transport; Oxidative stress; Phosphorylation; Proanthocyanidins - chemistry; Proanthocyanidins - pharmacology; Protein Transport - drug effects; RAW 264.7 Cells; Reactive Oxygen Species - metabolism; Science; Science (multidisciplinary); Signal Transduction; TLR4 protein; Toll-like receptors; Toll-Like Receptors - metabolism; Translocation; Tumor necrosis factor-α