JNK regulates muscle remodeling via myostatin/SMAD inhibition - Lessard, Sarah J.; MacDonald, Tara L.; Pathak, Prerana; Han, Myoung Sook; Coffey, Vernon G.; Edge, Johann; Rivas, Donato A.; Hirshman, Michael F.; Davis, Roger J.; Goodyear, Laurie J. (Nature communications, 2018)
Keywords: 13; 13/1; 13/106; 13/109; 13/95; 14; 14/34; 14/63; 38; 38/77; 631/80/86; 692/698/1671/1668; Adult; Animals; c-Jun protein; Cardiorespiratory fitness; Cell Nucleus - metabolism; Durability; Endurance; Enzyme Activation; Exercise; Fitness; Gene Expression Regulation; HEK293 Cells; Humanities and Social Sciences; Humans; Hypertrophy; Integrases - metabolism; JNK Mitogen-Activated Protein Kinases - metabolism; JNK protein; Mice, Inbred C57BL; Mice, Knockout; Molecular machines; multidisciplinary; Muscle Development; Muscle, Skeletal - metabolism; Muscle, Skeletal - pathology; Muscles; Muscles - metabolism; Musculoskeletal system; Myostatin; Myostatin - metabolism; Oxidation resistance; Phosphorylation; Physical Conditioning, Animal; Physical Endurance; Physical exercise; Protein Transport; Science; Science (multidisciplinary); Signal Transduction; Signaling; Skeletal muscle; Smad protein; Smad Proteins - antagonists & inhibitors; Smad Proteins - metabolism; Smad2 protein; Transcription factors