MYB96 recruits the HDA15 protein to suppress negative regulators of ABA signaling in Arabidopsis

• Published in: Nature communications Vol. 10; no. 1; p. 1713
• Main Authors: Lee, Hong Gil, Seo, Pil Joon
• Format: Journal Article
• Language: English
• Published: London Nature Publishing Group UK 12.04.2019; Nature Publishing Group; Nature Portfolio
• Subjects: 38; 38/1; 38/15; 38/35; 38/39; 38/88; 631/337/572/2102; 631/449/2661/2146; 631/449/2675; 64; 96; 96/95; Abscisic acid; Abscisic Acid - metabolism; Arabidopsis - genetics; Arabidopsis - metabolism; Arabidopsis Proteins - genetics; Arabidopsis Proteins - metabolism; Catalysis; Deficient mutant; Derepression; Down-regulation; Drought; Gene expression; Gene Expression Regulation, Plant; Genes; Genetic analysis; Genotype; Guanosine triphosphatases; Histone Deacetylases - genetics; Histone Deacetylases - metabolism; Histone H3; Histones - metabolism; Humanities and Social Sciences; Molecular modelling; multidisciplinary; Mutants; Mutation; Plants, Genetically Modified - metabolism; Promoter Regions, Genetic; Proteins; Regulators; Reproducibility of Results; Science; Science (multidisciplinary); Sensitivity; Signal Transduction; Signaling; Transcription activation; Transcription Factors - genetics; Transcription Factors - metabolism

Unlike activation of target genes in response to abscisic acid (ABA), how MYB96 transcription factor represses ABA-repressible genes to further enhance ABA responses remains unknown. Here, we show MYB96 interacts with the histone modifier HDA15 to suppress negative regulators of early ABA signaling....