P300-dependent STAT3 acetylation is necessary for angiotensin II-induced pro-fibrotic responses in renal tubular epithelial cells - Ni, Jun; Shen, Yang; Wang, Zhen; Shao, De-cui; Liu, Jia; Kong, Ya-li; Fu, Lan-jun; Zhou, Li; Xue, Hong; Huang, Yu; Zhang, Wei; Yu, Chen; et al. (Acta pharmacologica Sinica, 2014)
Keywords: Acetylation - drug effects; Angiotensin II - pharmacology; Animals; Biomedical and Life Sciences; Biomedicine; Cell Line; E1A-Associated p300 Protein - metabolism; Epithelial Cells - drug effects; Epithelial Cells - metabolism; Fibrosis - chemically induced; Fibrosis - metabolism; Immunology; Internal Medicine; Kidney Tubules - drug effects; Kidney Tubules - metabolism; Medical Microbiology; Original; original-article; P300; Pharmacology/Toxicology; Rats; STAT3; STAT3 Transcription Factor - metabolism; Vaccine; 乙酰化; 依赖性; 纤维化; 肾小管上皮细胞; 血管紧张素II; 诱导作用; p300; AG490; angiotensin II; STAT3; C646; renal fibrosis; renal tubular epithelial cells; JAK2; extracellular matrix; curcumin