APA (7th ed.) Citation

Wang, M., Han, J., Marcar, L., Black, J., Liu, Q., Li, X., . . . Willers, H. (2017). Radiation Resistance in KRAS-Mutated Lung Cancer Is Enabled by Stem-like Properties Mediated by an Osteopontin–EGFR Pathway. Cancer research (Chicago, Ill.), 77(8), 2018-2028. https://doi.org/10.1158/0008-5472.CAN-16-0808

Chicago Style (17th ed.) Citation

Wang, Meng, et al. "Radiation Resistance in KRAS-Mutated Lung Cancer Is Enabled by Stem-like Properties Mediated by an Osteopontin–EGFR Pathway." Cancer Research (Chicago, Ill.) 77, no. 8 (2017): 2018-2028. https://doi.org/10.1158/0008-5472.CAN-16-0808.

MLA (9th ed.) Citation

Wang, Meng, et al. "Radiation Resistance in KRAS-Mutated Lung Cancer Is Enabled by Stem-like Properties Mediated by an Osteopontin–EGFR Pathway." Cancer Research (Chicago, Ill.), vol. 77, no. 8, 2017, pp. 2018-2028, https://doi.org/10.1158/0008-5472.CAN-16-0808.