Exosomal miRNA-19b-3p of tubular epithelial cells promotes M1 macrophage activation in kidney injury - Lv, Lin-Li; Feng, Ye; Wu, Min; Wang, Bin; Li, Zuo-Lin; Zhong, Xin; Wu, Wei-Jun; Chen, Jun; Ni, Hai-Feng; Tang, Tao-Tao; Tang, Ri-Ning; Lan, Hui-Yao; et al. (Cell death and differentiation, 2020)
Keywords: 13/109; 13/51; 13/89; 14/1; 14/19; 14/28; 14/34; 38/61; 38/90; 42/41; 631/208/2490; 64/60; 692/420/256/2515; 692/699/1585; 96/21; Acute Kidney Injury - genetics; Acute Kidney Injury - metabolism; Adult; Aged; Animals; Apoptosis; Biochemistry; Biomedical and Life Sciences; Cell activation; Cell Biology; Cell Cycle Analysis; Cells, Cultured; Diabetic Nephropathies - urine; Diabetic nephropathy; Epithelial cells; Epithelial Cells - metabolism; Exosomes - genetics; Exosomes - metabolism; Female; Humans; Inflammation; Kidney - metabolism; Kidney - pathology; Kidney diseases; Kidney Tubules - metabolism; Life Sciences; Macrophage Activation; Macrophages; Macrophages - metabolism; Male; Mice; Mice, Inbred BALB C; Mice, Inbred C57BL; MicroRNAs; MicroRNAs - metabolism; MicroRNAs - urine; Middle Aged; miRNA; Nephritis - genetics; Nephritis - pathology; NF-kappa B - metabolism; Proteinuria - chemically induced; Proteinuria - genetics; Proteinuria - metabolism; RAW 264.7 Cells; Stem Cells; Suppressor of Cytokine Signaling 1 Protein - genetics; Suppressor of Cytokine Signaling 1 Protein - metabolism