Fenofibrate attenuates endothelial monocyte adhesion in chronic heart failure: an in vitro study - Huang, W. P.; Yin, W. H.; Chen, J. W.; Jen, H. L.; Young, M. S.; Lin, S. J. (European journal of clinical investigation, 2009)
Keywords: Aged; Aorta; Biological and medical sciences; Blotting, Western; Cardiology. Vascular system; Cell Adhesion - drug effects; Cell adhesion molecules; Chronic Disease; chronic heart failure; Endothelial cells; endothelial monocyte adhesion; Female; Fenofibrate; Fenofibrate - pharmacology; Gene expression; General aspects; Heart; Heart diseases; Heart Failure - pathology; Heart failure, cardiogenic pulmonary edema, cardiac enlargement; Humans; Inflammation; Inflammation - pathology; intercellular adhesion molecule 1; Leukocytes (mononuclear); Male; Medical sciences; Middle Aged; Monocytes; Monocytes - physiology; Peripheral blood mononuclear cells; peroxisome proliferator-activated receptor; Peroxisome proliferator-activated receptor-a; Tumor necrosis factor-a; vascular cell adhesion molecule 1; Vascular Cell Adhesion Molecule-1 - biosynthesis; Vascular Cell Adhesion Molecule-1 - drug effects; Vascular system; Heart failure; Endothelial cell; Monocyte; Fibrate derivatives; Cell adhesion molecule; endothelial monocyte adhesion; Cardiovascular disease; Inflammation; chronic heart failure; Adhesion; In vitro; Endothelium; Cell adhesion molecules; Fenofibrate; Medicine; Chronic; Heart disease; peroxisome proliferator-activated receptor; Peroxisome proliferator activated receptor; Antilipemic agent