NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart - Zhang, Hong; Xiao, Yang; Nederlof, Rianne; Bakker, Diane; Zhang, Pengbo; Girardin, Stephen E.; Hollmann, Markus W.; Weber, Nina C.; Houten, Sander M.; van Weeghel, Michel; Kibbey, Richard G.; Zuurbier, Coert J. (Frontiers in immunology, 2020)
Keywords: Animals; Biomarkers; Carbohydrate Metabolism; cardiac metabolism; Cytokines - metabolism; Disease Models, Animal; duration of ischemia; Gene Deletion; Glucose - metabolism; Immunology; infarct size; innate immunity; Mice; Mice, Knockout; Mitochondrial Proteins - genetics; Myocardial Reperfusion Injury - etiology; Myocardial Reperfusion Injury - metabolism; Myocardial Reperfusion Injury - pathology; Oxidation-Reduction; Oxidative Stress; oxygen consumption; Proto-Oncogene Proteins c-akt - metabolism; infarct size; duration of ischemia; innate immunity; cardiac metabolism; oxygen consumption