Protective effects of PEP-1-Catalase on stress-induced cellular toxicity and MPTP-induced Parkinson's disease - Eom, S.A., Hallym University, Chunchon, Republic of Korea; Kim, D.W., Gangnung-Wonju National University, Gangneung, Republic of Korea; Shin, M.J., Hallym University, Chunchon, Republic of Korea; Ahn, E.H., Hallym University, Chunchon, Republic of Korea; Chung, S.Y., Hallym University, Chunchon, Republic of Korea; Sohn, E.J., Hallym University, Chunchon, Republic of Korea; Jo, H.S., Hallym University, Chunchon, Republic of Korea; Jeon, S.J., Soonchunhyang University, Cheonan, Republic of Korea; Kim, D.S., Soonchunhyang University, Cheonan, Republic of Korea; Kwon, H.Y., Hallym University, Chunchon, Republic of Korea; Cho, S.W., University of Ulsan College of Medicine, Seoul, Republic of Korea; Han, K.H., Hallym University, Chunchon, Republic of Korea; et al. (BMB reports, 2015)
Keywords: 1-Methyl-4-phenyl-1,2,3,6-tetrahydropyridine; Animals; Brain - drug effects; Brain - metabolism; Cell Death - drug effects; Cell Line; Cell Survival - drug effects; Disease Models, Animal; DISEASES; Dopaminergic Neurons - drug effects; Dopaminergic Neurons - metabolism; Dopaminergic Neurons - pathology; ENFERMEDAD; Humans; MALADIES; Male; Mice, Inbred C57BL; Neuroprotective Agents - pharmacology; Neuroprotective Agents - therapeutic use; Parkinson Disease - drug therapy; Parkinson Disease - pathology; Parkinson's disease,ROS,PEP-1-Catalase,Protein therapy,Dopaminergic neuron; Recombinant Fusion Proteins - isolation & purification; Recombinant Fusion Proteins - pharmacology; Recombinant Fusion Proteins - therapeutic use; Signal Transduction - drug effects; Stress, Physiological - drug effects; Transduction, Genetic; 화학