Activation of the Nrf2-ARE Pathway Ameliorates Hyperglycemia-Mediated Mitochondrial Dysfunction in Podocytes Partly Through Sirt1 - Zhang, Qunzi; Deng, Qiongxia; Zhang, Jun; Ke, JianTing; Zhu, Ye; Wen, Ruo wei; Ye, Zengchun; Peng, Hui; Su, Zhong zhen; Wang, Cheng; Lou, Tanqi (Cellular physiology and biochemistry, 2018)
Keywords: Adenosine Triphosphate - metabolism; Animals; Antioxidant response element; Antioxidants; Cell Line; Creatinine - urine; Deoxyribonucleic acid; Diabetes; Diabetes mellitus; Diabetes Mellitus, Experimental - metabolism; Diabetes Mellitus, Experimental - pathology; DNA; Glucose - pharmacology; Humans; Hydroquinones - pharmacology; Hyperglycemia; Hyperglycemia - metabolism; Hyperglycemia - pathology; Inflammation; Injuries; Kidney diseases; Kinases; Male; Membrane Proteins - metabolism; Metabolism; Mice; Mitochondria; Mitochondria - drug effects; Mitochondria - metabolism; Mitochondrial DNA; NF-E2-Related Factor 2 - antagonists & inhibitors; NF-E2-Related Factor 2 - genetics; NF-E2-Related Factor 2 - metabolism; Original Paper; Oxidative stress; Permeability; Phosphorylation; Podocytes; Podocytes - cytology; Podocytes - drug effects; Podocytes - metabolism; Proteins; RNA Interference; RNA, Small Interfering - metabolism; Signal Transduction - drug effects; Sirt1; Sirtuin 1 - antagonists & inhibitors; Sirtuin 1 - genetics; Sirtuin 1 - metabolism; Superoxides - metabolism; Tert-butylhydroquinone; United States--US; Podocytes; Tert-butylhydroquinone; Sirt1; Antioxidant response element; Diabetes mellitus