Exogenous Tryptophan Promotes Cutaneous Wound Healing of Chronically Stressed Mice through Inhibition of TNF-α and IDO Activation - Bandeira, Luana Graziella; Bortolot, Beatriz Salari; Cecatto, Matheus Jorand; Monte-Alto-Costa, Andréa; Romana-Souza, Bruna (PloS one, 2015)
Keywords: AKT protein; Animals; Catabolism; Cell culture; Cell migration; Collagen; Collagen - genetics; Collagen - metabolism; Contraction; Cytokines; Dioxygenase; Embryology; Epinephrine; Euthanasia; Fibroblasts; Fibroblasts - drug effects; Fibroblasts - metabolism; Gene expression; Histology; Indoleamine-Pyrrole 2,3,-Dioxygenase - metabolism; Inflammation; Inflammatory response; Inhibition; Kinases; Laboratories; Lesions; Leukocyte migration; Leukocytes (neutrophilic); Lipid Peroxidation; Lymphocytes; Lymphocytes T; Macrophages; Macrophages - drug effects; Macrophages - metabolism; Male; Metanephrine - blood; Mice; Neutrophils - drug effects; Neutrophils - metabolism; Peroxidation; Phosphorylation; Proto-Oncogene Proteins c-akt - metabolism; Re-Epithelialization; Reduction; Rodents; Skin; Skin - drug effects; Skin - injuries; Skin - metabolism; Stress; Stress, Psychological - metabolism; Stress, Psychological - pathology; Stresses; Studies; Tryptophan; Tryptophan - blood; Tryptophan - pharmacology; Tumor Necrosis Factor-alpha - metabolism; Tumor necrosis factor-TNF; Tumor necrosis factor-α; Wound healing; Brazil; Rio de Janeiro Brazil