NanI and NanJ Sialidases of Clostridium perfringens Are Not Essential for Virulence - Chiarezza, Martina; Lyras, Dena; Pidot, Sacha J; Flores-Díaz, Marietta; Awad, Milena M; Kennedy, Catherine L; Cordner, Leanne M; Phumoonna, Tongted; Poon, Rachael; Hughes, Meredith L; Emmins, John J; Alape-Girón, Alberto; et al. (Infection and Immunity, 2009)
Keywords: Animal models; Animals; Bacterial Proteins; Bacterial Proteins - genetics; Bacterial Proteins - physiology; Biological and medical sciences; Cell Line, Tumor; Cell Survival; Clostridium; Clostridium perfringens; Clostridium perfringens - enzymology; Clostridium perfringens - pathogenicity; Cytotoxicity; enzymology; Extracellular enzymes; Fundamental and applied biological sciences. Psychology; Gas Gangrene; Gas Gangrene - microbiology; Gene Knockout Techniques; genes; genetics; homologous recombination; Melanoma; Mice; Mice, Inbred BALB C; Microbiology; Molecular Pathogenesis; Mutagenesis, Insertional; mutants; Mutation; Myonecrosis; Neuraminidase; Neuraminidase - genetics; Neuraminidase - physiology; pathogenicity; Pathogens; physiology; sialidase; Survival Analysis; Toxins; Virulence; Virulence Factors; Virulence Factors - genetics; Virulence Factors - physiology; Glycosylases; Microbiology; Enzyme; Glycosidases; Exo-α-sialidase; Virulence; Clostridiaceae; Clostridiales; Bacteria; Hydrolases; Immunity; Clostridium perfringens