Cardiomyocyte-Specific Deletion of the Vitamin D Receptor Gene Results in Cardiac Hypertrophy - Chen, Songcang; Law, Christopher S.; Grigsby, Christopher L.; Olsen, Keith; Hong, Ting-Ting; Zhang, Yan; Yeghiazarians, Yerem; Gardner, David G. (Circulation (New York, N.Y.), 2011)
Keywords: Animals; Biological and medical sciences; Blood and lymphatic vessels; Cardiology. Vascular system; Cardiomegaly - etiology; Cardiomegaly - genetics; Cardiomegaly - metabolism; Diseases of the peripheral vessels. Diseases of the vena cava. Miscellaneous; Gene Deletion; Gene Targeting; Heart; Heart failure, cardiogenic pulmonary edema, cardiac enlargement; Medical sciences; Mice; Mice, 129 Strain; Mice, Inbred C57BL; Mice, Knockout; Myocytes, Cardiac - metabolism; Myocytes, Cardiac - pathology; Receptors, Calcitriol - deficiency; Receptors, Calcitriol - genetics; Agonist; Isoprenaline; Bronchodilator; β2-Adrenergic receptor; β-Adrenergic receptor agonist; Cardiovascular disease; isoproterenol; β-Adrenergic receptor; gene deletion; Vitamin D; Deletion; vitamin D receptor; cardiac hypertrophy