CCL8 secreted by tumor-associated macrophages promotes invasion and stemness of glioblastoma cells via ERK1/2 signaling - Zhang, Xiang; Chen, Lu; Dang, Wei-qi; Cao, Mian-fu; Xiao, Jing-fang; Lv, Sheng-qing; Jiang, Wen-jie; Yao, Xiao-hong; Lu, Hui-min; Miao, Jing-ya; Wang, Yan; Yu, Shi-cang; et al. (Laboratory investigation, 2020)
Keywords: 14; 14/19; 14/63; 38/1; 38/61; 38/77; 631/250/98; 631/67/1922; 631/67/327; 631/67/580; 631/67/71; 64/60; 82/80; 96; 96/100; 96/21; 96/31; Animals; Antibodies; Brain - cytology; Brain - metabolism; Brain cancer; Brain Neoplasms - metabolism; Brain Neoplasms - pathology; CC chemokine receptors; CCR1 protein; CCR5 protein; Chemokine CCL8 - metabolism; Chemokines; Extracellular signal-regulated kinase; Glioblastoma; Glioblastoma - metabolism; Glioblastoma cells; Glioma; Glioma cells; Humans; Laboratory Medicine; Ligands; Macrophages; Macrophages - metabolism; MAP Kinase Signaling System - physiology; Medicine; Medicine & Public Health; Mice; Microenvironments; Neoplasm Invasiveness - physiopathology; Neoplastic Stem Cells - cytology; Pathology; Phosphorylation; Pseudopodia; Receptors; Tumor cells; Tumor Cells, Cultured; Tumors