The SARS-CoV-2 Omicron BA.1 spike G446S mutation potentiates antiviral T-cell recognition - Motozono, Chihiro; Toyoda, Mako; Tan, Toong Seng; Hamana, Hiroshi; Goto, Yoshihiko; Aritsu, Yoshiki; Miyashita, Yusuke; Oshiumi, Hiroyuki; Nakamura, Kimitoshi; Okada, Seiji; Udaka, Keiko; Kitamatsu, Mizuki; et al. (Nature communications, 2022)
Keywords: 13; 13/1; 13/31; 42; 42/109; 42/70; 45/22; 45/23; 45/44; 45/77; 631/250/1619/554; 631/250/254; 631/250/255/2514; 631/326/596/4130; 64/60; 82/80; 82/83; 96; Antibodies; Antibody response; Antigen presentation; Antigen processing; Antigens; Antiviral activity; Antiviral drugs; CD8 antigen; Cell activation; Cell recognition; COVID-19; Disease resistance; Epitopes; Histocompatibility antigen HLA; Humanities and Social Sciences; Immune response; Immune response (cell-mediated); Immune system; Lymphocytes; Lymphocytes T; multidisciplinary; Mutagenesis; Mutation; N-Terminus; Peptidase; Peptidases; Proteins; Recognition; Replication; Science; Science (multidisciplinary); Severe acute respiratory syndrome coronavirus 2; Spike protein; T cell receptors; Tripeptidylpeptidase II; Vaccines