Autophagy deficiency promotes triple-negative breast cancer resistance to T cell-mediated cytotoxicity by blocking tenascin-C degradation - Li, Zhi-Ling; Zhang, Hai-Liang; Huang, Yun; Huang, Jun-Hao; Sun, Peng; Zhou, Ning-Ning; Chen, Yu-Hong; Mai, Jia; Wang, Yan; Yu, Yan; Zhou, Li-Huan; Li, Xuan; et al. (Nature communications, 2020)
Keywords: 13/31; 13/51; 14/19; 38/109; 38/23; 38/35; 38/88; 38/89; 631/67/1347; 631/67/580/1884; 64/60; 82/58; 82/80; Anticancer properties; Autophagy; Biocompatibility; Breast cancer; CD8 antigen; Cell activation; Cytotoxicity; Degradation; Humanities and Social Sciences; Immune checkpoint inhibitors; Immunosuppression; Immunotherapy; Lymphocytes; Lymphocytes T; multidisciplinary; PD-1 protein; Phagocytosis; Science; Science (multidisciplinary); Skp2 protein; Tenascin; Tenascin C; Toxicity; Tumors