differential effects of single or repeated restraint stress on kainic acid-induced neuronal death in the hippocampal CA3 region: the role of glucocorticoid and various signal molecules - Kwon, Min-Soo; Seo, Young-Jun; Choi, Seung-Min; Choi, Hee-Woo; Jung, Jun-Sub; Park, Soo-Hyun; Suh, Hong-Won (Journal of neurochemistry, 2007)
Keywords: Ageing, cell death; Animals; Binding sites; Biochemistry; Biological and medical sciences; Cell Death; Cell Death - drug effects; Cell Death - physiology; Cell physiology; Dose-Response Relationship, Drug; drug effects; Fundamental and applied biological sciences. Psychology; Glucocorticoids; Glucocorticoids - physiology; Headache. Facial pains. Syncopes. Epilepsia. Intracranial hypertension. Brain oedema. Cerebral palsy; hippocampus; Hippocampus - drug effects; Hippocampus - metabolism; Hippocampus - pathology; Kainic Acid; Kainic Acid - toxicity; Male; Medical sciences; metabolism; methods; Mice; Mice, Inbred ICR; Molecular and cellular biology; Nervous system (semeiology, syndromes); Neurology; neuronal death; Neurons; Neurons - drug effects; Neurons - metabolism; Neurons - pathology; pathology; phosphorylated calcium/calmodulin dependent protein kinase II; phosphorylated cyclic-AMP response element binding protein; physiology; Proteins; Restraint, Physical; Restraint, Physical - methods; Signal Transduction; Signal Transduction - drug effects; Signal Transduction - physiology; Stress; Stress, Physiological; Stress, Physiological - metabolism; Stress, Physiological - pathology; Time Factors; toxicity; Phosphorylation; kainic acid; Toxicity; Central nervous system; Glucocorticoid; Stress; Encephalon; Treatment; Cell death; neuronal death; Glucocorticoid receptor; Serum; Antagonist; Transcription factor CREB; Hippocampus; phosphorylated cyclic-AMP response element binding protein; Biological receptor; phosphorylated calcium/calmodulin dependent protein kinase II