NAD(P)H: Quinone oxidoreductase 1 overexpression in hepatocellular carcinoma potentiates apoptosis evasion through regulating stabilization of X-linked inhibitor of apoptosis protein - Li, Wan-Yu; Zhou, Hong-Zhong; Chen, Yao; Cai, Xue-Fei; Tang, Hua; Ren, Ji-Hua; Wai Wong, Vincent Kam; Kwan Law, Betty Yuen; Chen, Yong; Cheng, Sheng-Tao; Yu, Hai-Bo; Cai, Hao-Yang; et al. (Cancer letters, 2019)
Keywords: Animals; Antioxidants; Apoptosis; Breast cancer; Carcinoma, Hepatocellular - enzymology; Carcinoma, Hepatocellular - pathology; Cell growth; Cell Line, Transformed; Cell proliferation; Colon; Gene expression; Hepatocellular carcinoma; Humans; Immunoglobulins; Kinases; Liver cancer; Liver Neoplasms - enzymology; Liver Neoplasms - pathology; Male; Mice; Mice, Inbred BALB C; Molecular modelling; NAD; NAD(P)H Dehydrogenase (Quinone) - genetics; NAD(P)H Dehydrogenase (Quinone) - metabolism; NQO1; Phosphorylation; Prostate cancer; Proteins; Quinone oxidoreductase; Skin cancer; Therapeutic applications; Tumorigenicity; X-linked inhibitor of apoptosis protein; X-Linked Inhibitor of Apoptosis Protein - metabolism; XIAP protein; United States--US; China; NQO1; Hepatocellular carcinoma; X-linked inhibitor of apoptosis protein; Apoptosis