Connexin 43 gap junctions contribute to brain endothelial barrier hyperpermeability in familial cerebral cavernous malformations type III by modulating tight junction structure - Johnson, Allison M; Roach, James P; Hu, Anna; Stamatovic, Svetlana M; Zochowski, Michal R; Keep, Richard F; Andjelkovic, Anuska V (The FASEB journal, 2018)
Keywords: Animals; Blood-Brain Barrier - metabolism; Blood-Brain Barrier - pathology; Cell Line; Claudin-5 - genetics; Claudin-5 - metabolism; Connexin 43 - genetics; Connexin 43 - metabolism; Disease Models, Animal; Endothelium, Vascular - metabolism; Endothelium, Vascular - pathology; Gap Junctions - genetics; Gap Junctions - metabolism; Gap Junctions - pathology; Hemangioma, Cavernous, Central Nervous System - genetics; Hemangioma, Cavernous, Central Nervous System - metabolism; Hemangioma, Cavernous, Central Nervous System - pathology; Humans; Mice; Mice, Knockout; Permeability; Tight Junctions - genetics; Tight Junctions - metabolism; Zonula Occludens-1 Protein - genetics; Zonula Occludens-1 Protein - metabolism; zonula occludens-1; PDCD10; cell-cell interaction; permeability