Vitamin D improves endothelial dysfunction and restores myeloid angiogenic cell function via reduced CXCL-10 expression in systemic lupus erythematosus - Reynolds, John A.; Haque, Sahena; Williamson, Kate; Ray, David W.; Alexander, M. Yvonne; Bruce, Ian N. (Scientific reports, 2016)
Keywords: 13/106; 13/51; 38/77; 692/308/575; 692/4019/592/16; 692/4023/1670/1613; Adult; Autoimmune diseases; Calcitriol - pharmacology; Cardiovascular diseases; Cell Adhesion - drug effects; Cell Count; Cell Differentiation - drug effects; Cell Movement - drug effects; Chemokine CXCL10 - metabolism; Down-Regulation - drug effects; Endothelium, Vascular - drug effects; Endothelium, Vascular - physiopathology; Health risks; Humanities and Social Sciences; Humans; Immunology; Interferons - metabolism; Lupus; Lupus Erythematosus, Systemic - metabolism; Lupus Erythematosus, Systemic - physiopathology; Middle Aged; Models, Biological; multidisciplinary; Myeloid Cells - drug effects; Myeloid Cells - pathology; Neovascularization, Physiologic - drug effects; Nitric Oxide Synthase Type III - metabolism; Phenotype; Science; Systemic lupus erythematosus; Vitamin D; Vitamin D - pharmacology