CXCL2, a new critical factor and therapeutic target for cardiovascular diseases

Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the physiological functions of cytoskeletal reorganization, cell migration, adhesion and immune responses. However, in the past decade, there have been st...

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Published inClinical and experimental hypertension (1993) Vol. 42; no. 5; pp. 428 - 437
Main Authors Guo, Lin-Ya, Yang, Fang, Peng, Li-Jun, Li, Yan-Bing, Wang, Ai-Ping
Format Journal Article
LanguageEnglish
Published England Taylor & Francis 03.07.2020
Taylor & Francis Group
Subjects
atherosclerosis
cardiovascular disease
CXCL2
inflammation
atherosclerosis
cardiovascular disease
inflammation
CXCL2
Online AccessGet full text
ISSN1064-1963
1525-6006
1525-6006
DOI10.1080/10641963.2019.1693585

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Abstract Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the physiological functions of cytoskeletal reorganization, cell migration, adhesion and immune responses. However, in the past decade, there have been studies showing that inflammatory chemokines play a key role in CVD. Importantly, CXC motif chemokine ligand 2 (CXCL2) has been shown to be involved in the pathogenesis of cardiovascular disease. CXCL2 exerts its effects on the cardiovascular system by mediating inflammatory responses, but the specific signaling pathways by which CXCL2 exerts its effects in CVD remain unknown and need to be further investigated. This review aims to investigate the expression changes of CXCL2 in acute myocardial infarction (AMI), atherosclerosis (AS), obesity, diabetes and ischemic stroke (IS) and its potential key role in cardiovascular disease in order to provide new ideas for the prevention and treatment of cardiovascular diseases.
AbstractList Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the physiological functions of cytoskeletal reorganization, cell migration, adhesion and immune responses. However, in the past decade, there have been studies showing that inflammatory chemokines play a key role in CVD. Importantly, CXC motif chemokine ligand 2 (CXCL2) has been shown to be involved in the pathogenesis of cardiovascular disease. CXCL2 exerts its effects on the cardiovascular system by mediating inflammatory responses, but the specific signaling pathways by which CXCL2 exerts its effects in CVD remain unknown and need to be further investigated. This review aims to investigate the expression changes of CXCL2 in acute myocardial infarction (AMI), atherosclerosis (AS), obesity, diabetes and ischemic stroke (IS) and its potential key role in cardiovascular disease in order to provide new ideas for the prevention and treatment of cardiovascular diseases.
Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the physiological functions of cytoskeletal reorganization, cell migration, adhesion and immune responses. However, in the past decade, there have been studies showing that inflammatory chemokines play a key role in CVD. Importantly, CXC motif chemokine ligand 2 (CXCL2) has been shown to be involved in the pathogenesis of cardiovascular disease. CXCL2 exerts its effects on the cardiovascular system by mediating inflammatory responses, but the specific signaling pathways by which CXCL2 exerts its effects in CVD remain unknown and need to be further investigated. This review aims to investigate the expression changes of CXCL2 in acute myocardial infarction (AMI), atherosclerosis (AS), obesity, diabetes and ischemic stroke (IS) and its potential key role in cardiovascular disease in order to provide new ideas for the prevention and treatment of cardiovascular diseases.Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the physiological functions of cytoskeletal reorganization, cell migration, adhesion and immune responses. However, in the past decade, there have been studies showing that inflammatory chemokines play a key role in CVD. Importantly, CXC motif chemokine ligand 2 (CXCL2) has been shown to be involved in the pathogenesis of cardiovascular disease. CXCL2 exerts its effects on the cardiovascular system by mediating inflammatory responses, but the specific signaling pathways by which CXCL2 exerts its effects in CVD remain unknown and need to be further investigated. This review aims to investigate the expression changes of CXCL2 in acute myocardial infarction (AMI), atherosclerosis (AS), obesity, diabetes and ischemic stroke (IS) and its potential key role in cardiovascular disease in order to provide new ideas for the prevention and treatment of cardiovascular diseases.
Author Guo, Lin-Ya
Wang, Ai-Ping
Li, Yan-Bing
Yang, Fang
Peng, Li-Jun
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Cites_doi 10.1161/ATVBAHA.114.303567
10.1677/erc.1.01301
10.1038/19546
10.2337/diabetes.52.11.2821
10.1002/ijc.28551
10.1146/annurev.cellbio.042308.113238
10.2337/dbi15-0013
10.1016/S0140-6736(12)61689-4
10.1007/s00018-011-0870-8
10.1111/bph.12246
10.1016/j.ijcard.2011.08.849
10.1038/ijo.2016.26
10.1248/bpb.33.1958
10.1084/jem.167.2.570
10.1074/jbc.M115.690081
10.1089/ars.2011.4487
10.1046/j.1523-1747.2000.00029.x
10.1007/s10585-015-9714-5
10.1126/science.aaa8064
10.1182/blood-2007-07-099648
10.1016/S1074-7613(00)80165-X
10.1161/01.CIR.0000148824.93600.F3
10.1006/jmcc.2001.1503
10.1096/fasebj.3.14.2687068
10.1080/13814788.2017.1398320
10.1371/journal.pone.0168562
10.1038/jcbfm.2012.198
10.1177/1060028018765939
10.15252/emmm.201404144
10.1097/00019052-200102000-00014
10.1007/s12603-018-1031-7
10.1161/CIRCRESAHA.111.261784
10.2174/157016112798829823
10.1016/j.biopha.2018.03.173
10.1161/CIRCRESAHA.114.302313
10.1124/pr.111.005074
10.1046/j.1471-4159.2002.01166.x
10.1038/nrcardio.2016.127
10.2741/3190
10.1160/TH08-12-0837
10.4049/jimmunol.165.9.5269
10.1161/ATVBAHA.110.206011
10.1016/j.molimm.2003.07.001
10.1172/JCI67604
10.1016/j.jacc.2011.08.034
10.1089/ars.2006.8.1907
10.1016/j.cardiores.2006.09.011
10.1161/ATVBAHA.114.305218
10.1161/CIRCRESAHA.110.218420
10.2337/db11-0853
10.1161/ATVBAHA.114.303585
10.1080/19381980.2016.1267078
10.2741/2853
10.1038/nri3399
10.1016/j.molimm.2016.11.007
10.1038/sj.jid.5700842
10.1371/journal.pone.0066515
10.4049/jimmunol.1700129
10.1038/86286
10.1016/j.cardiores.2006.11.030
10.1016/j.hoc.2013.11.010
10.1182/blood-2013-02-486217
10.7326/M18-0222
10.1097/FJC.0b013e318241c385
10.1155/2013/610950
10.4049/jimmunol.0903843
10.1002/ana.23876
10.1182/blood-2004-08-3193
10.1016/j.ijcard.2009.09.548
10.1177/0022034518755697
10.4103/1995-705X.137493
10.1038/nm.4068
10.1152/ajpendo.00347.2013
10.1046/j.1365-2796.2001.00867.x
10.1530/JOE-16-0501
10.3727/096368911X627372
10.1073/pnas.90.19.8992
10.1073/pnas.87.19.7732
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References CIT0072
CIT0071
CIT0030
CIT0074
CIT0073
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CIT0076
CIT0031
CIT0075
CIT0034
CIT0078
CIT0033
CIT0077
CIT0070
CIT0036
CIT0035
CIT0079
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CIT0037
CIT0039
CIT0041
CIT0040
CIT0043
CIT0042
CIT0001
CIT0045
CIT0044
CIT0080
CIT0003
CIT0047
CIT0046
CIT0005
CIT0049
CIT0004
CIT0048
CIT0007
CIT0006
CIT0009
CIT0008
CIT0050
CIT0052
CIT0051
CIT0010
CIT0054
CIT0053
CIT0012
CIT0056
CIT0011
CIT0055
Huss R (CIT0013) 2004; 88
Polak M (CIT0002) 2018; 11
CIT0014
CIT0058
CIT0057
CIT0016
CIT0015
CIT0059
CIT0018
CIT0017
CIT0019
CIT0061
CIT0060
CIT0063
CIT0062
CIT0021
CIT0065
CIT0020
CIT0064
CIT0023
CIT0067
CIT0022
CIT0066
CIT0025
CIT0069
CIT0024
CIT0068
CIT0027
CIT0026
CIT0029
CIT0028
References_xml – ident: CIT0063
  doi: 10.1161/ATVBAHA.114.303567
– ident: CIT0024
  doi: 10.1677/erc.1.01301
– ident: CIT0042
  doi: 10.1038/19546
– ident: CIT0058
  doi: 10.2337/diabetes.52.11.2821
– ident: CIT0026
  doi: 10.1002/ijc.28551
– ident: CIT0044
  doi: 10.1146/annurev.cellbio.042308.113238
– ident: CIT0052
  doi: 10.2337/dbi15-0013
– ident: CIT0001
  doi: 10.1016/S0140-6736(12)61689-4
– ident: CIT0019
  doi: 10.1007/s00018-011-0870-8
– ident: CIT0047
  doi: 10.1111/bph.12246
– ident: CIT0062
  doi: 10.1016/j.ijcard.2011.08.849
– ident: CIT0056
  doi: 10.1038/ijo.2016.26
– ident: CIT0075
  doi: 10.1248/bpb.33.1958
– ident: CIT0016
  doi: 10.1084/jem.167.2.570
– ident: CIT0034
  doi: 10.1074/jbc.M115.690081
– ident: CIT0070
  doi: 10.1089/ars.2011.4487
– ident: CIT0059
  doi: 10.1046/j.1523-1747.2000.00029.x
– ident: CIT0025
  doi: 10.1007/s10585-015-9714-5
– ident: CIT0040
  doi: 10.1126/science.aaa8064
– ident: CIT0021
  doi: 10.1182/blood-2007-07-099648
– ident: CIT0027
  doi: 10.1016/S1074-7613(00)80165-X
– ident: CIT0080
  doi: 10.1161/01.CIR.0000148824.93600.F3
– ident: CIT0065
  doi: 10.1006/jmcc.2001.1503
– ident: CIT0017
  doi: 10.1096/fasebj.3.14.2687068
– ident: CIT0003
  doi: 10.1080/13814788.2017.1398320
– ident: CIT0060
  doi: 10.1371/journal.pone.0168562
– ident: CIT0077
  doi: 10.1038/jcbfm.2012.198
– volume: 88
  start-page: 170
  year: 2004
  ident: CIT0013
  publication-title: Verh Dtsch Ges Pathol
– ident: CIT0007
  doi: 10.1177/1060028018765939
– ident: CIT0051
  doi: 10.15252/emmm.201404144
– ident: CIT0076
  doi: 10.1097/00019052-200102000-00014
– ident: CIT0038
  doi: 10.1007/s12603-018-1031-7
– ident: CIT0041
  doi: 10.1161/CIRCRESAHA.111.261784
– ident: CIT0008
  doi: 10.2174/157016112798829823
– ident: CIT0037
  doi: 10.1016/j.biopha.2018.03.173
– ident: CIT0049
  doi: 10.1161/CIRCRESAHA.114.302313
– ident: CIT0014
  doi: 10.1124/pr.111.005074
– ident: CIT0078
  doi: 10.1046/j.1471-4159.2002.01166.x
– ident: CIT0004
  doi: 10.1038/nrcardio.2016.127
– ident: CIT0015
  doi: 10.2741/3190
– ident: CIT0064
  doi: 10.1160/TH08-12-0837
– ident: CIT0072
  doi: 10.4049/jimmunol.165.9.5269
– ident: CIT0079
  doi: 10.1161/ATVBAHA.110.206011
– ident: CIT0023
  doi: 10.1016/j.molimm.2003.07.001
– ident: CIT0039
  doi: 10.1172/JCI67604
– ident: CIT0069
  doi: 10.1016/j.jacc.2011.08.034
– ident: CIT0067
  doi: 10.1089/ars.2006.8.1907
– ident: CIT0071
  doi: 10.1016/j.cardiores.2006.09.011
– ident: CIT0048
  doi: 10.1161/ATVBAHA.114.305218
– ident: CIT0032
  doi: 10.1161/CIRCRESAHA.110.218420
– ident: CIT0012
  doi: 10.2337/db11-0853
– ident: CIT0045
  doi: 10.1161/ATVBAHA.114.303585
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  doi: 10.1080/19381980.2016.1267078
– ident: CIT0018
  doi: 10.2741/2853
– ident: CIT0033
  doi: 10.1038/nri3399
– ident: CIT0035
  doi: 10.1016/j.molimm.2016.11.007
– ident: CIT0057
  doi: 10.1038/sj.jid.5700842
– ident: CIT0061
  doi: 10.1371/journal.pone.0066515
– ident: CIT0028
  doi: 10.4049/jimmunol.1700129
– volume: 11
  start-page: 0022
  issue: 7
  year: 2018
  ident: CIT0002
  publication-title: Kardiol Pol
– ident: CIT0005
  doi: 10.1038/86286
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  doi: 10.1016/j.cardiores.2006.11.030
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  doi: 10.1182/blood-2013-02-486217
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  doi: 10.7326/M18-0222
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  doi: 10.1097/FJC.0b013e318241c385
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  doi: 10.1155/2013/610950
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  doi: 10.4049/jimmunol.0903843
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  doi: 10.1002/ana.23876
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  doi: 10.1182/blood-2004-08-3193
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Snippet Cardiovascular diseases (CVDs) are the leading cause of death worldwide. Previous studies have shown that inflammatory chemokines are involved in the...
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SubjectTerms atherosclerosis
cardiovascular disease
CXCL2
inflammation
Title CXCL2, a new critical factor and therapeutic target for cardiovascular diseases
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