A Mutation in the Herpes Simplex Virus Type 1 (HSV-1) UL29 Gene is Associated with Anti-Herpesvirus Drugs’ Susceptibility - Yamada, Souichi; Harada, Shizuko; Fujii, Hikaru; Kinoshita, Hitomi; Nguyen, Phu Hoang Anh; Shibamura, Miho; Yoshikawa, Tomoki; Kawahara, Madoka; Ebihara, Hideki; Saijo, Masayuki; Fukushi, Shuetsu (Viruses, 2024)
Keywords: Acyclovir; Acyclovir - pharmacology; Acyclovir - therapeutic use; Animals; Antiviral Agents - pharmacology; Antiviral Agents - therapeutic use; Antiviral drugs; Chemicals; Chlorocebus aethiops; Cloning; Disease resistance; DNA-Binding Proteins - genetics; DNA-Binding Proteins - metabolism; DNA-directed DNA polymerase; DNA-Directed DNA Polymerase - genetics; DNA-Directed DNA Polymerase - metabolism; Drug resistance; Drug resistance in microorganisms; Drug Resistance, Viral - genetics; E coli; Gene mutations; Genes; Genes, Viral; Genetic aspects; Genomes; Health aspects; Herpes simplex; Herpes Simplex - drug therapy; Herpes Simplex - virology; Herpes simplex virus; herpes simplex virus type 1 (HSV-1); Herpes viruses; Herpesvirus 1, Human - drug effects; Herpesvirus 1, Human - genetics; Humans; ICP8 protein; Infections; Kinases; Microbial Sensitivity Tests; Missense mutation; Mutagenesis; Mutants; Mutation; Mutation, Missense; Penicillin; Polyclonal antibodies; Proteins; Thymidine kinase; Thymidine Kinase - genetics; Thymidine Kinase - metabolism; UL29; Varicella; Vero Cells; Viral Proteins - genetics; Viral Proteins - metabolism; Whole genome sequencing; Japan; St Louis Missouri; United Kingdom--UK; United States--US; Germany; UL29; acyclovir; drug resistance; herpes simplex virus type 1 (HSV-1)