Prostate cancer cell-derived exosomes ZNF667-AS1 reduces TGFBR1 mRNA stability to inhibit Treg expansion and DTX resistance by binding to U2AF1 - Shi, Zhenfeng; Pu, Wenjing; Li, Min; Aihemaitijiang, Mierzhayiti; Li, Shuo; Zhang, Xiaoan; Liu, Bide; Sun, Min; Li, Jiuzhi; Li, Zhiwei (Molecular medicine (Cambridge, Mass.), 2024)
Keywords: Androgens; Animals; Apoptosis; Cancer therapies; Cell growth; Cell Line, Tumor; Cell Proliferation; Chemotherapy; Docetaxel; Docetaxel - pharmacology; Drug Resistance, Neoplasm - genetics; Exosomes; Exosomes - metabolism; Flow cytometry; Gene Expression Regulation, Neoplastic; Humans; Lymphocytes; Male; Mice; Prostate cancer; Prostatic Neoplasms - genetics; Prostatic Neoplasms - metabolism; Prostatic Neoplasms - pathology; Receptor, Transforming Growth Factor-beta Type I - genetics; Receptor, Transforming Growth Factor-beta Type I - metabolism; RNA Stability; RNA, Long Noncoding - genetics; RNA, Long Noncoding - metabolism; RNA, Messenger - genetics; RNA, Messenger - metabolism; T-Lymphocytes, Regulatory - immunology; T-Lymphocytes, Regulatory - metabolism; Tregs; ZNF667-AS1; United States--US; China; Xinjiang China; Exosomes; Prostate cancer; Tregs; Docetaxel; ZNF667-AS1