Interleukin 1B proinflammatory genotypes protect against gastro-oesophageal reflux disease through induction of corpus atrophy - Ando, T (Gut, 2006)
Keywords: Adult; Biological and medical sciences; Esophagitis; Esophagitis - genetics; Esophagitis - microbiology; Esophagus; Female; Gastritis, Atrophic; Gastritis, Atrophic - complications; Gastritis, Atrophic - genetics; Gastritis, Atrophic - microbiology; Gastroenterology. Liver. Pancreas. Abdomen; Gastroesophageal Reflux; Gastroesophageal Reflux - genetics; Gastroesophageal Reflux - microbiology; Gastroesophageal Reflux - prevention & control; Genetic Predisposition to Disease; Genotype; Helicobacter Infections; Helicobacter Infections - complications; Helicobacter pylori; Humans; Interleukin-1; Interleukin-1 - genetics; Interleukin-10; Interleukin-10 - genetics; Male; Medical sciences; Middle Aged; Other diseases. Semiology; Polymerase Chain Reaction; Polymerase Chain Reaction - methods; Prospective Studies; Severity of Illness Index; Tumor Necrosis Factor-alpha; Tumor Necrosis Factor-alpha - genetics; Atrophy; Gastroesophageal reflux; Microbiological investigation; Typing; Interleukin; Esophageal disease; Gastroenterology; Digestive diseases; Genotype