Adipocyte deletion of the oxygen-sensor PHD2 sustains elevated energy expenditure at thermoneutrality - Wang, Rongling; Gomez Salazar, Mario; Pruñonosa Cervera, Iris; Coutts, Amanda; French, Karen; Pinto, Marlene Magalhaes; Gohlke, Sabrina; García-Martín, Ruben; Blüher, Matthias; Schofield, Christopher J.; Kourtzelis, Ioannis; Stimson, Roland H.; et al. (Nature communications, 2024)
Keywords: 13; 13/106; 13/31; 13/51; 38; 38/39; 631/154/53; 631/443/319; 692/308/1426; 82/80; Adipocytes; Adipocytes - metabolism; Adipocytes, Brown - metabolism; Adipose tissue; Adipose tissue (brown); Adipose Tissue, Brown - metabolism; Adult; Animals; Basic Helix-Loop-Helix Transcription Factors - genetics; Basic Helix-Loop-Helix Transcription Factors - metabolism; Biomarkers; Body fat; Burning; Dormancy; Energy expenditure; Energy Metabolism; Female; Humanities and Social Sciences; Humans; Hypoxia; Hypoxia-Inducible Factor-Proline Dioxygenases - genetics; Hypoxia-Inducible Factor-Proline Dioxygenases - metabolism; Hypoxia-inducible factors; Living conditions; Male; Metabolic disorders; Mice; Mice, Inbred C57BL; Mice, Knockout; Middle Aged; multidisciplinary; Oxygen; Oxygen - metabolism; Prolyl hydroxylase; Promoter Regions, Genetic; Proteomics; Science; Science (multidisciplinary); Strategy; Thermogenesis - genetics; Uncoupling Protein 1 - genetics; Uncoupling Protein 1 - metabolism