Downregulation of tumor suppressor RACK1 by Helicobacter pylori infection promotes gastric carcinogenesis through the integrin β-1/NF-κB signaling pathway - Hu, Yi; Liu, Jian-Ping; Li, Xue-Yang; Cai, Yan; He, Cong; Li, Nian-Shuang; Xie, Chuan; Xiong, Zhi-Juan; Ge, Zhong-Ming; Lu, Nong-Hua; Zhu, Yin (Cancer letters, 2019)
Keywords: Animals; Antigens, Bacterial - metabolism; Bacterial Proteins - metabolism; Cancer; Carcinogenesis; Cell cycle; Cell Line, Tumor; Cytokines; Disease control; Disease prevention; Down-Regulation; Gastric cancer; Gerbillinae; Helicobacter Infections - metabolism; Helicobacter pylori; Helicobacter pylori - metabolism; Humans; Infections; Inflammation; Integrin beta1 - metabolism; Integrin β-1; Kinases; Male; Medical research; Molecular modelling; Neoplasm Proteins - genetics; Neoplasm Proteins - metabolism; NF-kappa B - metabolism; NF-κB; NF-κB protein; Pathogens; Prognosis; Protein kinase C; Proteins; RACK1; Receptors for Activated C Kinase - genetics; Receptors for Activated C Kinase - metabolism; Signal Transduction; Stomach Neoplasms - genetics; Stomach Neoplasms - metabolism; Stomach Neoplasms - microbiology; Tumor suppressor genes; Beijing China; United States--US; China; RACK1; NF-κB; Helicobacter pylori; Gastric cancer; Integrin β-1