Mediator release after nasal aspirin provocation supports different phenotypes in subjects with hypersensitivity reactions to NSAIDs - Campo, P.; Ayuso, P.; Salas, M.; Plaza, M. C.; Cornejo‐García, J. A.; Doña, I.; Torres, M. J.; Blanca‐López, N.; Canto, G.; Guéant, J.‐L.; Sanak, M.; Blanca, M. (Allergy (Copenhagen), 2013)
Keywords: Administration, Intranasal; Adolescent; Adult; Aged; Allergies; Angioedema - chemically induced; Angioedema - genetics; Angioedema - immunology; Anti-Inflammatory Agents, Non-Steroidal - adverse effects; Aspirin - administration & dosage; Aspirin - adverse effects; aspirin‐exacerbated cutaneous disease; Female; Genotype & phenotype; Humans; Immunophenotyping - methods; Inflammation Mediators - metabolism; inflammatory mediators; Life Sciences; lysine nasal challenge; Male; Middle Aged; multiple nonsteroidal anti‐inflammatory drug‐induced urticaria/angioedema; Nonsteroidal anti-inflammatory drugs; Respiratory Hypersensitivity - chemically induced; Respiratory Hypersensitivity - genetics; Respiratory Hypersensitivity - immunology; Urticaria - chemically induced; Urticaria - genetics; Urticaria - immunology; Young Adult; inflammatory mediators; nonsteroidal anti-inflammatory drugs; aspirin-exacerbated cutaneous disease; lysine nasal challenge; multiple nonsteroidal anti-inflammatory drug-induced urticaria/angioedema